HCSGD entry for MUC7


1. General information

Official gene symbolMUC7
Entrez ID4589
Gene full namemucin 7, secreted
Other gene symbolsMG2
Links to Entrez GeneLinks to Entrez Gene

2. Neighbors in the network

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This gene isn't in Literature mining network.

3. Gene ontology annotation

GO ID

GO term

Evidence

Category

GO:0005515Protein bindingIPImolecular_function
GO:0005576Extracellular regionIEAcellular_component
GO:0005796Golgi lumenTAScellular_component
GO:0016266O-glycan processingTASbiological_process
GO:0043687Post-translational protein modificationTASbiological_process
GO:0044267Cellular protein metabolic processTASbiological_process
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4. Expression levels in datasets

  • Meta-analysis result

p-value upp-value downFDR upFDR down
0.72010187680.90897021390.99999024731.0000000000

  • Individual experiment result
    ( "-" represent NA in the specific microarray platform )

Data sourceUp or downLog fold change
GSE11954Up0.0055289764
GSE13712_SHEARUp0.0374685025
GSE13712_STATICUp0.1189280609
GSE19018Up0.1317418001
GSE19899_A1Up0.0400433456
GSE19899_A2Down-0.0349230996
PubMed_21979375_A1Up0.1230017154
PubMed_21979375_A2Down-0.0740533648
GSE35957Down-0.0428225596
GSE36640Up0.0342551907
GSE54402Up0.0773905808
GSE9593Down-0.0354390562
GSE43922Up0.0132977262
GSE24585Down-0.0987791945
GSE37065Up0.0057376474
GSE28863_A1Up0.0497639865
GSE28863_A2Up0.2161202889
GSE28863_A3Up0.2145820257
GSE28863_A4Up0.0231268406
GSE48662Down-0.0112153864

5. Regulation relationships with compounds/drugs/microRNAs

  • Compounds

Not regulated by compounds

  • Drugs

Not regulated by drugs

  • MicroRNAs

  • mirTarBase

MiRNA_name

mirBase ID

miRTarBase ID

Experiment

Support type

References (Pubmed ID)

hsa-miR-26b-5pMIMAT0000083MIRT029062MicroarrayFunctional MTI (Weak)19088304
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  • mirRecord
No target information from mirRecord

6. Text-mining results about the gene

Gene occurances in abstracts of cellular senescence-associated articles: 2 abstracts the gene occurs.


PubMed ID of the article

Sentenece the gene occurs

3000548There was no significant age-dependent change in the ability to synthesize PtdIns4P and PtdIns(4,5)P2 or in the response of the PtdIns and PtdIns4P kinases to Mg2+
378256Ouabain and anoxia have no effect on K+ transport mechanisms; in contrast, both processes are completely blocked by dicyclohexylcarbodiimide, an inhibitor of the Mg2+ -dependent ATPase activity
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